Overview of Sinus Rhythms and Arrhythmias
This lecture provides an in-depth explanation of sinus rhythms and various arrhythmias originating from the sinus node and atrioventricular (AV) junction. It emphasizes the importance of ECG interpretation to differentiate these rhythms for timely clinical intervention.
Understanding Arrhythmias and the Sinus Node
- Arrhythmia Definition: Abnormal cardiac rhythm.
- Sinus Node: Primary pacemaker located in the right atrium, generating impulses that spread through atrial muscle.
- Control: Autonomic nervous system regulates sinus node discharge rate.
- Sinus Node Dysfunction: Caused by fibrosis, ischemia, or degeneration, common in elderly, may cause dizziness, palpitations, or syncope. For more on this, see our Comprehensive Guide to Patient Identification and Normal ECG Interpretation.
- Management: Depends on symptoms; may include medication or pacemaker implantation.
Subsidiary Pacemakers and Their Role
- When the sinus node fails, subsidiary pacemakers in the AV node and ventricles initiate impulses at slower rates:
- Sinus node: 60-200 bpm
- AV node: 40-60 bpm
- Ventricles: 20-40 bpm
Sinus Rhythm and Its Identification on ECG
- Defined by impulse initiation in the sinus node.
- ECG Characteristics:
- Positive P waves in leads II, III, aVF
- Negative P wave in aVR
- Biphasic P wave in V1
Sinus Bradycardia
- Heart rate <60 bpm.
- Causes include high vagal tone (athletes), hypothyroidism, drugs, ischemia, sinus node fibrosis. For a deeper understanding of ECG features, refer to our Comprehensive Guide to ECG Waveforms, Intervals, and Heart Rate Calculation.
- ECG Features:
- P wave precedes QRS complex with fixed PR interval.
- Differentiated from junctional rhythm by presence of upright P waves.
Sinus Tachycardia
- Heart rate >100 bpm.
- Causes include exercise, fever, anxiety, anemia, thyrotoxicosis.
- ECG Features:
- P wave precedes QRS complex.
- Rate calculation example shows 150 bpm.
Junctional Rhythms
- Originate from AV junction (area around AV node and bundle of His).
- Rate typically 40-60 bpm (escape rhythm), 60-100 bpm (accelerated), or >100 bpm (tachycardia). For more on junctional rhythms, see our Comprehensive Guide to Heart Conduction and ECG Fundamentals.
- ECG Characteristics:
- Narrow QRS complexes.
- P waves may be inverted, appear before, during, or after QRS, or be hidden.
- Examples:
- Junctional escape rhythm: rate ~60 bpm, hidden P waves.
- Accelerated junctional rhythm: rate 60-100 bpm, inverted P waves with short PR.
- Junctional tachycardia: rate >100 bpm, inverted P waves after QRS.
Sinus Pause, Sinus Arrest, and Sinoatrial Exit Block
- Sinus Pause: Failure of SA node to generate impulse; pause duration >1.5 seconds but not exact multiple of preceding PP interval.
- Sinus Arrest: Pause >3 seconds.
- Sinoatrial Exit Block: Intermittent failure of impulse conduction from SA node to atria; pause is exact multiple of preceding PP interval.
SA Exit Block Degrees
- First Degree: Prolonged conduction time, not visible on ECG.
- Second Degree:
- Mobitz Type 1: Progressive conduction delay with intermittent missed beats; pause less than twice shortest PP interval.
- Mobitz Type 2: Fixed PP intervals with sudden missed beats; pause is multiple of PP interval.
- Third Degree: Complete block with no P waves and long pauses, leading to ectopic rhythms.
Summary
- The heart's electrical activation normally starts at the SA node.
- Dysfunction leads to subsidiary pacemaker activity at slower rates.
- Sinus rhythm is identified by characteristic P waves and rate.
- Bradycardia and tachycardia are defined by heart rate thresholds and have distinct ECG features.
- Junctional rhythms differ by P wave morphology and timing. For a comprehensive understanding of these rhythms, check out our Understanding Cardiac Electrophysiology and Arrhythmias: Key ECG Insights.
- Sinus pauses, arrests, and SA exit blocks have specific ECG patterns critical for diagnosis.
- Early recognition and differentiation of these rhythms are vital for appropriate management and prevention of complications.
[Music] [Music] in this lecture video i am going to
discuss about sinus rhythms and braid arrhythmias i am going to discuss about sinus rhythm
sinus bradycardia sinus tachycardia and braid arrhythmias at the end of the lecture video you will
be able to identify sinus rhythms and how to differentiate sinus rhythm from junctional rhythms and how to identify
and differentiate between sinus pass sinus arrest and cyanoatrial exit block what is an arrhythmia an abnormality in
cardiac rhythm is called an arrhythmia sa node is the predominant pacemaker of the heart and it depolarizes
spontaneously it is a modified cardiac muscle situated at the superior part of lateral wall of
right atrium the fibers of a sa node are continuous with fibers of atrial muscle so once the
impulses are generated it spreads rapidly through the atrial muscle the rate of sinus node discharge is
controlled by autonomic nervous system there are conditions which lead to sinus node dysfunction such as fibrosis
degenerative condition or ischemia of the sinus node this leads to variety of arrhythmias
commonly found in elderly and they may be asymptomatic or present with symptoms such as dizziness palpitation or
syncopal attack management depends on the patient's symptoms and may be by drugs to improve
the heart rate or with permanent pacemakers the point i want to stress upon here is
it is vital to differentiate these rhythm abnormalities based on the ecg and to intervene early to prevent any
life threatening conditions next point i am going to discuss is what happens if the sa node is dysfunctional
or suppressed and what are subsidiary pacemakers and what is their role
if sa node is dysfunctional or suppressed in order to continue hot beating other subsidiary pacemakers in
the av node specialized conducting system and muscle may initiate electrical activation
but typically subsidiary pacemakers discharge at a slower rate these are the various pacemaker size and
rate of depolarization sa node depolarizes at a rate of 6200 beats per minute
av node depolarizes at a rate of 40 to 60 bits per minute and ventricles at a rate of 20 to 40 beats per minute
before discussing about sinus bradycardia and sinus tachycardia let me explain about sinus rhythm
what is sinus rhythm if the depolarization begins in the sa node of the heart it is said to be in sinus
rhythm in the ecg p wave characterized by positive p waves in lead 2 3 avf
negative p wave in avr and biphasic p wave in v1 next about bradycardia and how to
differentiate sinus bradycardia with junctional rhythms so bradycardia is defined as heart rate
less than 60 bits per minute in adults during faithful state bradycardia results from either failure
of impulse initiation or impulse conduction depressed automaticity leads to failure
of impulse initiation asymptomatic bradycardia can be seen in people with high vagal tone such as in athletes so
it is vital to differentiate bradycardia as whether physiological or pathological to intervene accordingly
so there are many causes for bradycardia there are some extrinsic causes and intrinsic causes extrinsic causes such
as hypothyroidism drugs etc and intrinsic causes are ischemia and information of the sinus node
fibrosis of the sinus node or six sinus syndrome bradycardia may be due to sinus
bradycardia junctional rhythms or atrioventricular blocks
now how to recognize sinus bradycardia and how to differentiate sinus bradycardia from junctional rhythm
so in this ecg let us first calculate the rate to do that first trace the
r waves so here is an r wave here is an r wave in between there are 1 2 3 4 5 6
6 big boxes so 300 divided by 6 is equal to 50 the rate here is less than 60 so it is bradycardia
now how to say sinus there is a p wave which precedes the qrs complex
and here is a p wave which precedes the qrs complex and there is a fixed pr
interval so to say sinus
there is an upright p wave present in the qrs complex and
followed by a p wave preceding a qrs complex so this is an
example of sinus bradycardia if there is no p wave present in the qrs complex then it is called junctional
rhythm now moving on to sinus tachycardia when the sinus rate is accelerated more
than 100 beats per minute then it is called sinus tachycardia sinus tachycardia may be due to intrinsic
sinus node abnormalities such as enhanced automaticity or due to abnormal autonomic regulation of the heart such
as enhanced sympathetic tone or depressed parasympathetic tone it is a secondary phenomenon and
underlying causes to be investigation there can be acute or chronic causes acute causes such as exercise pain
anxiety fever acute heart failure pulmonary embolism etc and chronic causes such as anemia
thyrotoxicosis catecholamine excess etc so in this ecg let us calculate the rate here so this
is an r wave this is an r wave in between there are one two three four five six seven eight nine
there are nine small boxes so let us take ten thousand five 1500 divided by 10 is equal to 150 the rate
here is more than 100 so it is called tachycardia now how to say sinus there is a p wave which precedes the qrs
complex followed by a t wave then a p wave which precedes the qrs complex followed by a t wave so this is an
example of sinus tachycardia then moving on to junctional rhythm junctional dysrhythmias originate in the
av junction there is area around av node and bundle of his junctional rhythm is identified based on
the rate which is usually less that is around 40 to 60 bits per minute as it originates from other than sinus node
based on the qrs width which is usually normal as it follows the usual conduction system and importantly based
on the morphology of p waves this point i want to stress upon again that sinus p waves in lead 2 is upright
or positive and junctional p waves may be inverted which comes before or after the qrs complex or it may be a hidden p
wave this picture explains about normal sinus conduction and about junctional
conduction so based on the sequence of depolarization there are several
possible outcomes so these are some of the complexes here in that we can see various p wave
morphologies so this is a p wave morphology which is negative which comes before the qrs
complex and there is shortened pr interval and here you cannot able to find a p
wave and it is a hidden p wave here and in this complex you can see a p wave which comes after the qrs complex
so this p wave which is negative and before the qrs complex indicates that atria is depolarized from bottom up
and the hidden p wave indicates that atria and ventricles are depolarized simultaneously
and p wave which comes after the qrs complex indicates that av node initiated depolarization
but ventricles depolarized just before the atria now i am going to discuss about
junctional rhythms such as junctional escape rhythm accelerated junctional rhythm as well as
junctional tachycardia junctional escape rhythm arises from av junction at a rate of 40 to 60 beats per minute
it is characterized by heart rate around 40 to 60 bits per minute rhythm which is regular and p
waves which are inverted may appear before during or after the qrs complex and qrs complexes are relatively narrow
as is it usually follows the usual conduction system if p wave is present pure intervals will
be shorter than normal let us take this example first let us calculate the rate here is an r wave
here is an orbit in between there are one two three four five there are five big boxes so 300 divided by phi is equal
to 60 the rate here is 60 and the rhythm is a regular rhythm and you cannot able to see a p wave here
so there is a hidden p wave in this ecg and the qrs is normal as it follows the
usual conduction system this is an example of junctional escape rhythm
next about accelerated junctional rhythm which arises from the av junction at a rate of 60 to 100 bits per minute
it is due to increased automaticity of the av node or decreased automaticity of the sa node which is characterized by
rate around 60 to 100 beats per minute a regular rhythm p waves
may be inverted appear before during or after the qrs complex qrs
complexes are relatively narrow and p waves if present and the pure interval will be shorter than normal
let us take this example this is an r wave over here this is an r wave over here there are one two three
big boxes so 300 divided by 3 is equal to 100 so the rate is 100 the rhythm is a regular rhythm
and p wave which comes before the qrs complex and it is inverted and there is a shorter pr
and the qrs complex is normal as it usually follows the normal conduction system this is an example of accelerated
junctional rhythm next about junctional tachycardia it is a fast ectopic rhythm
arises from bundle office at a rate more than 100 bits per minute it is characterized by rate more than 100 bits
per minute regular rhythm p waves are inverted may appear before during or after the qrs
complex and qrs complex is normal in this example you can see r wave here and an r wave here
and you can see one two two big boxes between two r waves so 300 divided by 2 is equal to 150 the rate is more than
100 and the rhythm is a regular rhythm and if you see the p wave it has come
after the cures complex and the cures complex is normal this is an example of junctional tachycardia
next i am going to discuss about sinus pass sinus arrest and how to differentiate
sinus pulse sinus arrest from sa exit block sinus node consists of p cells or
pacemaker cells which forms the impulses and t cells which transmit the impulses to the atrial
muscle sinus pass is the failure of atrial activation due to inability of sa node
to form impulses this resulted in pass without visible p waves
the differentiating point between sinus pass versus cynital exit block is that the
pass is not an exact multiple of preceding pp interval sinus pass is characterized by absence
of p waves for a duration of more than 1.5 seconds if the delay is more than 3 seconds then
it is called sinus arrest let us take this example
in this ecg there is a pass
and if you take this pp interval preceding pp intervals
the pass is not an exact multiple of preceding pp interval
so if we calculate the number of boxes between the r waves so 1
2 3 4 5 6 7 8 9 10 around 10 big boxes so 10 into 0.2 is equal to 2 seconds so the delay is
around 2 seconds so this is an example of sinus pass
seen in normal individuals with increased vegal tone hypersensitive corrected sinus drugs such as digitalis
and inferior wall myocardial infarction next about cyanoatrial exit block as the name indicates cynital exit block
is due to intermittent failure in conduction of sinus impulses into atria the differentiating point between
cyanoatrial exit block from sinus pass is that the pass here is an exact multiple of preceding pp interval
cyanital exit block is graded into three degrees first degree sa block second degree sa
block and third degree sa block first degree assay block in this time for propagation of sinus impulse into
atria is significantly prolonged this cannot be detected on a surface ecg let us take this example
top one is normal ecg and below is first degree sa block if you compare the arrow marks
of both ecgs instead of atrial depolarization over here it has been delayed
so there is a delay in atrial depolarization this is an example of first degree sa block
second degree xa block is further classified as mobitz type 1sa block and mobit's type 2 assay block
mobility's type 1 assay block is characterized by progressive prolongation of sa conduction with
intermittent failure of impulses to conduct to surrounding atrial tissue no p waves appear for one beat and the
sequence begins again there is a phenomenon called group beating that means grouping of pqrs
complex with progressively shortened pp interval followed by a pass
and the pass is less than twice the shortest cycle if we take this as an example
this is a pp interval and compared to this pp interval that is
around 23 small boxes next pp interval is around 21 small boxes and followed by a
pass and then the sequence begins again so this pass is less than twice the
shortest pp interval so this is an example of mobitz type 1 block
mobitz type 2 block is characterized by fixed pp interval there is no change in sa node conduction
before and after the pass the pass is multiple of
preceding pp interval let us take this example so
compared to this pp interval this pp interval is
equal followed by a pass over here and the pause is an exact multiple of
the preceding pp interval so this is an example of
mobitz type 2 essay block third degree or complete sa block which is manifest as no p waves with long
pauses leading to ectopic atrial or ventricular rhythms so if you see there is a p wave over
here and next p wave comes over here so there is a long pass over here this is an example of
third degree or complete essay block summary electrical activation of the heart normally originates in the sa node
if sa node is dysfunctional or suppressed other subsidiary pacemakers may initiate electrical activation
subsidiary pacemakers discharge at a slower rate if the depolarization begins in the sa
node of the heart it is said to begin sinus rhythm bradycard is defined as heart rate less
than 60 bits per minute and results from either failure of impulse initiation or impulse conduction
tachycardia occurs when the heart rate is more than 100 bits per minute junctional rhythm originates in the av
junction and it is identified based on the rate qrs width and morphology of p waves
junctional p waves can be inverted p wave and it comes before or after the qrs complex or it may be a hidden pva
sinus pause or arrest is the failure of say no to discharge sa exit block
is due to intermittent failure in conduction of sinus symptoms into atria sa block is graded into three degrees
first degree is a block second degree assay block which is further classified into type one mobitz block and type two
mobitz block and third degree is a block these are my references thank you
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